Clostridium difficile infection and fecal bacteriotherapy: Difference between revisions
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[[Image:gene.png|thumb|right|Figure 4, The five genes endcoding toxin A and B as well as three regulatory gene products lie on a 19.6 KB pathogenicity locus. (Voth and Ballard 2005).]] | [[Image:gene.png|thumb|right|Figure 4, The five genes endcoding toxin A and B as well as three regulatory gene products lie on a 19.6 KB pathogenicity locus. (Voth and Ballard 2005).]] | ||
[[Image: | [[Image:spores.png|thumb|right|Figure 3. C. difficile spore exposure causes germination in hosts with certain primary bile salts such as taurocholate. After germination, vegetative cells grow and release toxins into the human host. Starvation induces sporulation and spores are spread through feces. Image courtesy of Seekatz and Young (2014).]] | ||
Revision as of 03:43, 21 April 2015
Introduction
By Rebecca Varnell
Introduce the topic of your paper. What microorganisms are of interest? Habitat? Applications for medicine and/or environment?
Section 1
Include some current research, with at least one figure showing data.
Section 2
Include some current research, with at least one figure showing data.
Section 3
Include some current research, with at least one figure showing data.
References
[1] Nazarko, L. (2015). Infection control: Clostridium difficile. British Journal Of Healthcare Assistants, 9(1), 20-25.