Group B Strep and Pregnancy: Difference between revisions
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<br>By Shawn Ruiz<br> | <br>By Shawn Ruiz<br> | ||
<br>Group B Strep (GBS), also known as Streptococcus agalactiae, is a Gram-positive, beta-hemolytic, catalase-negative, facultative anaerobe that is a normal component of the gastrointestinal and genitourinary tracts<ref name=aa>[https://en.wikipedia.org/wiki/Streptococcus_agalactiae]</ref>. In fact, GBS colonizes the gastrointestinal and genitourinary tracts of up to 50% of healthy adults<ref name=bb>[https://pubmed.ncbi.nlm.nih.gov/17088932/]</ref>. Most healthy adults who are colonized by GBS will not experience any symptoms or GBS-related infections. While the bacteria is usually harmless in healthy adults, it is a major cause of meningitis, pneumonia, and and sepsis in neonates<ref name=cc>[https://evidencebasedbirth.com/groupbstrep/]</ref>. Moreover, GBS is the leading infectious cause of mortality in neonates in the United States<ref name=dd>[https://www.ncbi.nlm.nih.gov/books/NBK482443/#:~:text=Preterm%20infants%20with%20early%2Donset,in%20term%20infants%5B2%5D.]</ref>. GBS causes both early onset and late onset infections in neonates, which occur during and after the first week of a neonate's life, respectively<ref name=dd/>. While early and late onset GBS infections in neonates are possible, current interventions are only effective in preventing early onset GBS infections<ref name=dd/>. The main risk factor for an early-onset GBS infection in a neonate is colonization of a birthing person's genital tract with Group B strep during labor<ref name=dd/>. | <br>Group B Strep (GBS), also known as Streptococcus agalactiae, is a Gram-positive, beta-hemolytic, catalase-negative, facultative anaerobe that is a normal component of the gastrointestinal and genitourinary tracts<ref name=aa>[https://en.wikipedia.org/wiki/Streptococcus_agalactiae]</ref>. In fact, GBS colonizes the gastrointestinal and genitourinary tracts of up to 50% of healthy adults<ref name=bb>[https://pubmed.ncbi.nlm.nih.gov/17088932/]</ref>. Most healthy adults who are colonized by GBS will not experience any symptoms or GBS-related infections. While the bacteria is usually harmless in healthy adults, it is a major cause of meningitis, pneumonia, and and sepsis in neonates<ref name=cc>[https://evidencebasedbirth.com/groupbstrep/]</ref>. Moreover, GBS is the leading infectious cause of mortality in neonates in the United States; between four and six percent of babies who develop GBS disease die<ref name=dd>[https://www.ncbi.nlm.nih.gov/books/NBK482443/#:~:text=Preterm%20infants%20with%20early%2Donset,in%20term%20infants%5B2%5D.]</ref><ref name=ee>[https://en.wikipedia.org/wiki/Streptococcus_agalactiae]</ref>. GBS causes both early onset and late onset infections in neonates, which occur during and after the first week of a neonate's life, respectively<ref name=dd/>. While early and late onset GBS infections in neonates are possible, current interventions are only effective in preventing early onset GBS infections<ref name=dd/>. The main risk factor for an early-onset GBS infection in a neonate is colonization of a birthing person's genital tract with Group B strep during labor<ref name=dd/>. | ||
Revision as of 01:46, 15 March 2021
Introduction
By Shawn Ruiz
Group B Strep (GBS), also known as Streptococcus agalactiae, is a Gram-positive, beta-hemolytic, catalase-negative, facultative anaerobe that is a normal component of the gastrointestinal and genitourinary tracts[1]. In fact, GBS colonizes the gastrointestinal and genitourinary tracts of up to 50% of healthy adults[2]. Most healthy adults who are colonized by GBS will not experience any symptoms or GBS-related infections. While the bacteria is usually harmless in healthy adults, it is a major cause of meningitis, pneumonia, and and sepsis in neonates[3]. Moreover, GBS is the leading infectious cause of mortality in neonates in the United States; between four and six percent of babies who develop GBS disease die[4][5]. GBS causes both early onset and late onset infections in neonates, which occur during and after the first week of a neonate's life, respectively[4]. While early and late onset GBS infections in neonates are possible, current interventions are only effective in preventing early onset GBS infections[4]. The main risk factor for an early-onset GBS infection in a neonate is colonization of a birthing person's genital tract with Group B strep during labor[4].
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Authored for BIOL 238 Microbiology, taught by Joan Slonczewski, 2021, Kenyon College.