Sarcoptes scabiei: Difference between revisions

From MicrobeWiki, the student-edited microbiology resource
No edit summary
Line 20: Line 20:
==Pathogenesis==
==Pathogenesis==
===Transmission===
===Transmission===
<i>Sarcoptes scabiei</i> is transmitted via person to person contact. Usually prolonged contact is required for scabies to spread. Consequently, household members and significant others are the significant mode of transmission. The most common source of infection in adults is from sexual partners. Scabies may be spread via inanimate objects such as clothing and bedding but the chances are significantly low because scabies do not survive for more than two-three days without human contact. If the symptoms of scabies have become sever enough to cause crusted scabies, transmission via clothing and other articles increases significantly.


===Infectious dose, incubation, and colonization===
===Infectious dose, incubation, and colonization===

Revision as of 12:39, 24 July 2014

This is a curated page. Report corrections to Microbewiki.
Microbiology in Italy[1]

Etiology/Bacteriology

Taxonomy

| Domain = Animalia | Phylum = Eumetazoa | Class = Arachnida | Order = Astigmata | Family = Sarcoptidae | Genus = [[Sarcoptes]] | species = [[Sarcoptes scabies]]

NCBI: Taxonomy Genome: []

Description

Pathogenesis

Transmission

Sarcoptes scabiei is transmitted via person to person contact. Usually prolonged contact is required for scabies to spread. Consequently, household members and significant others are the significant mode of transmission. The most common source of infection in adults is from sexual partners. Scabies may be spread via inanimate objects such as clothing and bedding but the chances are significantly low because scabies do not survive for more than two-three days without human contact. If the symptoms of scabies have become sever enough to cause crusted scabies, transmission via clothing and other articles increases significantly.

Infectious dose, incubation, and colonization

Epidemiology

Virulence factors

Clinical features

Diagnosis

Treatment

Prevention

Host Immune Response

References