Wohlfahritiimonas chitiniclastica: Difference between revisions
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==Description and significance== | ==Description and significance== | ||
Wohlfahrtiimonas chitiniclastica is a Gram-negative, aerobic bacterium of the class Gammaproteobacteria [[#References|[3]]]. It was first identified in 2008 from parasitic fly (Wohlfahrtia magnifica) larvae as a species within a new genus, Wohlfahrtiimonas [[#References|[3]]]. To date, three species within the genus have been identified, with W. chitiniclastica being the only one involved in human infection [[#References|[4]]]. W. chitiniclastica is a serious pest of livestock, and has been linked to myiasis, a condition where fly larvae infest living and necrotic tissue, particularly in wound infections caused by W. magnifica larvae [[#References|[5][6][10]]]. Myiasis caused by W. chitiniclastica is particularly severe in people with chronic wounds, poor sanitation, or who are immunocompromised [[#References|[7][8]]]. From its first isolation in 2008 to 2025, 44 cases of W. chitiniclastica infection have been reported globally [[#References|[4 | Wohlfahrtiimonas chitiniclastica is a Gram-negative, aerobic bacterium of the class Gammaproteobacteria [[#References|[3]]]. It was first identified in 2008 from parasitic fly (Wohlfahrtia magnifica) larvae as a species within a new genus, Wohlfahrtiimonas [[#References|[3]]]. To date, three species within the genus have been identified, with W. chitiniclastica being the only one involved in human infection [[#References|[4]]]. W. chitiniclastica is a serious pest of livestock, and has been linked to myiasis, a condition where fly larvae infest living and necrotic tissue, particularly in wound infections caused by W. magnifica larvae [[#References|[5][6][10]]]. Myiasis caused by W. chitiniclastica is particularly severe in people with chronic wounds, poor sanitation, or who are immunocompromised [[#References|[7][8]]]. From its first isolation in 2008 to 2025, 44 cases of W. chitiniclastica infection have been reported globally [[#References|[4]]]. From these previous case studies and current research, the genome structure, cell structure, basic metabolic processes, and virulence factors of W. chitiniclastica have been identified. Despite increasing clinical reports, the exact mechanism of pathogenesis and transmission routes by W. chitiniclastica remains unclear, thus inhibiting proper prevention and treatment [[#References|[9]]]. | ||
==Genome structure== | ==Genome structure== | ||
Revision as of 19:58, 8 December 2025
Classification
Higher order taxa
Domain: Bacteria [1][2].
Kingdom: Pseudomonadati [1][2].
Phylum: Pseudomonadota (Proteobacteria) [1][2].
Class: Gammaproteobacteria (g-proteobacteria) [3].
Order: Cardiobacteriales [1][2].
Family: Igantzschineriaceae [1][2].
Genus: Wohlfahrtiimonas [3].
Species: Wohlfahrtiimonas chitiniclastica [3].
Description and significance
Wohlfahrtiimonas chitiniclastica is a Gram-negative, aerobic bacterium of the class Gammaproteobacteria [3]. It was first identified in 2008 from parasitic fly (Wohlfahrtia magnifica) larvae as a species within a new genus, Wohlfahrtiimonas [3]. To date, three species within the genus have been identified, with W. chitiniclastica being the only one involved in human infection [4]. W. chitiniclastica is a serious pest of livestock, and has been linked to myiasis, a condition where fly larvae infest living and necrotic tissue, particularly in wound infections caused by W. magnifica larvae [5][6][10]. Myiasis caused by W. chitiniclastica is particularly severe in people with chronic wounds, poor sanitation, or who are immunocompromised [7][8]. From its first isolation in 2008 to 2025, 44 cases of W. chitiniclastica infection have been reported globally [4]. From these previous case studies and current research, the genome structure, cell structure, basic metabolic processes, and virulence factors of W. chitiniclastica have been identified. Despite increasing clinical reports, the exact mechanism of pathogenesis and transmission routes by W. chitiniclastica remains unclear, thus inhibiting proper prevention and treatment [9].
Genome structure
Cell structure
Metabolic processes
Ecology
Pathology
Current Research
References
It is required that you add at least five primary research articles (in same format as the sample reference below) that corresponds to the info that you added to this page. [Sample reference] Faller, A., and Schleifer, K. "Modified Oxidase and Benzidine Tests for Separation of Staphylococci from Micrococci". Journal of Clinical Microbiology. 1981. Volume 13. p. 1031-1035.
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for [http://www.bu.edu/academics/cas/courses/cas-bi-311/ BI 311 General
Microbiology], 2024, Boston University.
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