Zoonosis: Brucellosis in Animals and Humans: Difference between revisions
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<br> [[Image:BTP.jpg|thumb|300px|right| <b>Figure 2.</b> The various transmission pathways of <i>B. melitensis</i>. Pasteurization, fermentation, exposure to sunlight, and infection of certain paratenic hosts such as horses or humans are considered dead ends in that they either kill or prevent significant transmission of <i>Brucella</i>. <ref>[https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4026726/. Retrospective and prospective perspectives on zoonotic brucellosis. 2014. Moreno, E. <i>Front Microbiol.</i> 5: 213.]</ref>]] | <br> [[Image:BTP.jpg|thumb|300px|right| <b>Figure 2.</b> The various transmission pathways of <i>B. melitensis</i>. Pasteurization, fermentation, exposure to sunlight, and infection of certain paratenic hosts such as horses or humans are considered dead ends in that they either kill or prevent significant transmission of <i>Brucella</i>. <ref>[https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4026726/. Retrospective and prospective perspectives on zoonotic brucellosis. 2014. Moreno, E. <i>Front Microbiol.</i> 5: 213.]</ref>]] | ||
<br> [[Image:bdist.png|thumb|500px|right|<b>Figure | <br> [[Image:BTP.jpg|thumb|300px|right| <b>Figure 3.</b> The phylogeny of the genus <i>Brucella</i> aligned with the phylogeny of their respective, preferred host species. The width of the cones in the <i>Brucella</i> phylogeny is proportional to the number of strains analyzed for that species. The host phylogeny is represented in millions of years. <ref>[https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4026726/. Retrospective and prospective perspectives on zoonotic brucellosis. 2014. Moreno, E. <i>Front Microbiol.</i> 5: 213.]</ref>]] | ||
<br> [[Image:bdist.png|thumb|500px|right|<b>Figure 4.</b> Global occurrences of Brucellosis in humans. The highest incidences are recorded in Syria (1603.4 incidences per 1M people per year), Mongolia (605.9 incidences per 1M people per year), and Kyrgyzstan (362.2 incidences per 1M people per year). The incidence of Brucellosis in the United States is 0.4 per 1M people per year. <ref>[http://journals.plos.org/plosmedicine/article?id=10.1371/journal.pmed.0040317#pmed-0040317-g001 Ariza, J., Bosilkovski, M., Cascio, A., Colmenero, J. D. <i>et al.</i> "Perspectives for the Treatment of Brucellosis in the 21st Century: The Ioannina Recommendations." 2008. PLoS Med 4(12): e317]</ref>]] | |||
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Revision as of 02:00, 22 April 2017
Introduction
By Hannah Wedig
Figure 2. The various transmission pathways of B. melitensis. Pasteurization, fermentation, exposure to sunlight, and infection of certain paratenic hosts such as horses or humans are considered dead ends in that they either kill or prevent significant transmission of Brucella. [3]
Figure 3. The phylogeny of the genus Brucella aligned with the phylogeny of their respective, preferred host species. The width of the cones in the Brucella phylogeny is proportional to the number of strains analyzed for that species. The host phylogeny is represented in millions of years. [4]
Figure 4. Global occurrences of Brucellosis in humans. The highest incidences are recorded in Syria (1603.4 incidences per 1M people per year), Mongolia (605.9 incidences per 1M people per year), and Kyrgyzstan (362.2 incidences per 1M people per year). The incidence of Brucellosis in the United States is 0.4 per 1M people per year. [5]
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References
- ↑ "Brucella Melitensis." Database of Bacterial Food Pathogen. Birla Institute of Scientific Research, 2015.
- ↑ Brucellosis in terrestrial wildlife. 2013. Godfroid, J., Garin-Bastuji, B., Saegerman, C., Blasco, J. M. Rev. sci. tech. Off. int. Epiz. 32 (1): 27-42
- ↑ Retrospective and prospective perspectives on zoonotic brucellosis. 2014. Moreno, E. Front Microbiol. 5: 213.
- ↑ Retrospective and prospective perspectives on zoonotic brucellosis. 2014. Moreno, E. Front Microbiol. 5: 213.
- ↑ Ariza, J., Bosilkovski, M., Cascio, A., Colmenero, J. D. et al. "Perspectives for the Treatment of Brucellosis in the 21st Century: The Ioannina Recommendations." 2008. PLoS Med 4(12): e317
- ↑ Hodgkin, J. and Partridge, F.A. "Caenorhabditis elegans meets microsporidia: the nematode killers from Paris." 2008. PLoS Biology 6:2634-2637.
- ↑ Bartlett et al.: Oncolytic viruses as therapeutic cancer vaccines. Molecular Cancer 2013 12:103.
Authored for BIOL 238 Microbiology, taught by Joan Slonczewski, 2017, Kenyon College.
