Talk:Microbial Infection of Burn Wounds
In the second to last paragraph of the introduction, a few things could be clarified. Explaining “necrosis due to decreased perfusion” would be helpful, as would explaining what the eschar is. In the section where P. aeruginosa is discussed, it looks like there’s a typo in the title in the spelling of aeruginosa. Also, in the treatment section discussion of honey, you refer to S. aeruginosa- I’m guessing this should be either S. aureus or P. aeruginosa. Overall though it’s interesting and well-done.
After reading the topic on biofilm creation on teeth I thought this topic complement the topic. In ways it did. How P. aeraginosa uses quorum sensing to invade the burn wound is similar to how microbe on the teeth use communication to colonize the biofilm. Also it was similar in that the environment is first inhabited by gram-positive bacteria then gram-negative. It is incredible how quickly infection of burn woulnds can occur. It makes sense though because of exposure to air and it allows microbes such as Staphyloccus aureus to become opportunistic. It is interesting the types of prevention they use such as honey and bacteriophages to treat antiobiotic resistance in burn wounds. With the advancement in medicine today hopefully new and more effective means of treatment will be available. Just one question: Can adhesion proteins be a drug target?