Human Chromosomal Integration of Latent State Human Herpes Virus 6 (HHV-6): Difference between revisions
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==Pathophysiology of HHV-6 Infection== | ==Pathophysiology of HHV-6 Infection== | ||
[[Image:Rash.jpg|thumb|400px| | [[Image:Rash.jpg|thumb|400px|left| [http://andbabybmakesthree.wordpress.com/2009/11/11/wordless-wednesday-roseola/ Figure 4.] HHV-6 infection results in roseola, often diagnosed by a characteristic red rash. The roseola rash can form as either raised bumps or can be flat. It is non-contagious and is usually found on the neck, abdomen, back, and trunk, but can also be present on the arms and legs]] | ||
===Initial Infection=== | ===Initial Infection=== |
Revision as of 20:13, 1 November 2010
By: Kerri-Lynn Conrad
Introduction
Genomic Structure of HHV-6
HHV-6 Replication Cycle
Epidemiology
Pathophysiology of HHV-6 Infection
Initial Infection
Latency in Healthy Children and Adults
Reactivation in Immunosuppressed Individuals
HHV-6 and Associated Disease States
Latency through Human Chromosomal Integration
Transmission through Germ Line
Future Work
References
Edited by Kerri-Lynn Conrad, student of Joan Slonczewski for BIOL 375 Virology, 2010, Kenyon College.