Helicobacter pylori as a causative agent of Gastric Cancer: Difference between revisions
From MicrobeWiki, the student-edited microbiology resource
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== | ==<i>Helicobacter pylori</i>Description and Genome== | ||
== | ==Gastric Cancer== | ||
== | ==Symptoms and Treatment== | ||
==Process of Infection== | |||
==Adhesin== | |||
==DNA Repair Pathways== | |||
==Inflammation== | |||
==Future Work== | |||
==References== | ==References== |
Revision as of 17:37, 22 April 2015
Introduction
File:Https://gi.jhsps.org/Upload/200802291540 18983 000.jpg
Figure 2. Hematoxylin and eosin (H&E) stained histological section of the stomach mucosa indicating colonization of H. pylori. Courtesy of Johns Hopkins Medicine.
File:Http://www.nature.com/nrc/journal/v4/n9/images/nrc1433-f1.jpg
Figure 3. Induction of Cytoxin-associated gene A (CagA) gene by H. Pylori as a mechanism of cellular and DNA regulatory inhibition.
File:Http://www.pubcan.org/images/large/Fig 4-04 A.jpg
Figure 4. Advanced Gastric Carcinoma.
File:Http://upload.wikimedia.org/wikipedia/commons/9/9a/H pylori virulence factors en.png
Figure 5. H. pylori virulence factors.
Other examples:
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Subscript: H2O
Superscript: Fe3+
Helicobacter pyloriDescription and Genome
Gastric Cancer
Symptoms and Treatment
Process of Infection
Adhesin
DNA Repair Pathways
Inflammation
Future Work
References
[Sample reference]
Edited by Alexandra Kruse, student of Joan Slonczewski for BIOL 238 Microbiology, 2015, Kenyon College.