Helicobacter pylori as a causative agent of Gastric Cancer: Difference between revisions

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==Section 1==
==<i>Helicobacter pylori</i>Description and Genome==
Include some current research, with at least one image.<br><br>


==Section 2==
==Gastric Cancer==
Include some current research, with at least one image.<br><br>


==Conclusion==
==Symptoms and Treatment==
Overall text length should be at least 1,000 words (before counting references), with at least 2 images. Include at least 5 references under Reference section.<br><br>
 
==Process of Infection==
 
==Adhesin==
 
==DNA Repair Pathways==
 
==Inflammation==
 
==Future Work==


==References==
==References==

Revision as of 17:37, 22 April 2015

Introduction

Figure 1. Transmission electron micrograph image of Helicobacter pylori taken by Dr. Yutaka Tsutsumi, Department of Pathology, Fujita Health University School of Medicine
File:Https://gi.jhsps.org/Upload/200802291540 18983 000.jpg
Figure 2. Hematoxylin and eosin (H&E) stained histological section of the stomach mucosa indicating colonization of H. pylori. Courtesy of Johns Hopkins Medicine.
File:Http://www.nature.com/nrc/journal/v4/n9/images/nrc1433-f1.jpg
Figure 3. Induction of Cytoxin-associated gene A (CagA) gene by H. Pylori as a mechanism of cellular and DNA regulatory inhibition.
File:Http://www.pubcan.org/images/large/Fig 4-04 A.jpg
Figure 4. Advanced Gastric Carcinoma.



Other examples:
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Subscript: H2O
Superscript: Fe3+




Helicobacter pyloriDescription and Genome

Gastric Cancer

Symptoms and Treatment

Process of Infection

Adhesin

DNA Repair Pathways

Inflammation

Future Work

References

[Sample reference]


Edited by Alexandra Kruse, student of Joan Slonczewski for BIOL 238 Microbiology, 2015, Kenyon College.